@article {tb63,
	title = {Para-influenza 2 virus infections in adult volunteers},
	journal = {The Journal of Hygiene},
	volume = {61},
	year = {1963},
	pages = {4},
	author = {Taylor-Robinson, D. and Bynoe, M. L.}
}
@article {bargellini2011parameters,
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	volume = {45},
	number = {6},
	year = {2011},
	pages = {2315{\textendash}2321},
	publisher = {Elsevier},
	author = {Bargellini, Annalisa and Marchesi, Isabella and Righi, Elena and Ferrari, Angela and Cencetti, Stefano and Borella, Paola and Rovesti, Sergio}
}
@article {792,
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	year = {2011},
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	author = {CDC}
}
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	year = {2022},
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	volume = {74},
	year = {2000},
	pages = {2},
	author = {Brett, P. J. and Woods, D. E.}
}
@article {dupont_pathogenesis_1971,
	title = {Pathogenesis of {Escherichia} coli {Diarrhea}},
	journal = {New England Journal of Medicine},
	volume = {285},
	number = {1},
	year = {1971},
	note = {Publisher: Massachusetts Medical Society},
	pages = {1{\textendash}9},
	issn = {0028-4793},
	doi = {10.1056/NEJM197107012850101},
	url = {https://www.nejm.org/doi/10.1056/NEJM197107012850101},
	author = {DuPont, Herbert L. and Formal, Samuel B. and Hornick, Richard B. and Snyder, Merrill J. and Libonati, Joseph P. and Sheahan, Daniel G. and LaBrec, Eugene H. and Kalas, John P.}
}
@article {dfhslslk71,
	title = {Pathogenesis of Escherichia coli diarrhea},
	journal = {The New England journal of medicine},
	volume = {285},
	year = {1971},
	pages = {1},
	author = {DuPont, H L and Formal, S B and Hornick, R B and Snyder, M J and Libonati, J P and Sheahan, D G and LaBrec, E H and Kalas, J P}
}
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	year = {1963},
	pages = {61-66},
	author = {Suptel, E A}
}
@article {jshr82,
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	journal = {Infection and Immunity},
	volume = {37},
	year = {1982},
	pages = {2},
	url = {http://iai.asm.org/content/37/2/771.abstract},
	author = {Jahrling, P B and Smith, S and Hesse, R A and Rhoderick, J B}
}
@article {levine_pathogenesis_1973,
	title = {Pathogenesis of {Shigella} dysenteriae 1 ({Shiga}) {Dysentery}},
	journal = {The Journal of Infectious Diseases},
	volume = {127},
	number = {3},
	year = {1973},
	pages = {261{\textendash}270},
	abstract = {The Shiga bacillus is exceptional among enteric pathogens in possessing two potential pathogenetic modes of action, invasiveness and enterotoxin. The role of each was investigated. ,Two fully virulent (invasive toxigenic) and two modified (noninvasive toxigenic and invasive nontoxigenic) Shiga strains were defined in animal and in-vitro models. The virulent strains produced disease in volunteers in doses as low as 101 organisms. Illness was characterized by fever, short-lived diarrhea, and passage of scanty stools of blood and mucus. During illness, the pathogen was isolated from one of 16 samples of small-intestinal contents, and free toxin could not be demonstrated. Concomitantly, large numbers of Shiga organisms were excreted in stool. The major lesions were in the large bowel where clinical colitis was confirmed by proctoscopy and biopsy. Large numbers of a noninvasive toxigenic strain (106-1011 organisms) were well tolerated by 85 of 86 men. One volunteer had dysentery after the organism had reverted to an invasive form. In contrast, an invasive nontoxigenic strain caused shigellosis in monkeys and volunteers. Epithelial penetration is the cardinal virulence property of Shigella dysenteriae 1. The role of Shiga toxin in human disease remains to be defined.},
	issn = {0022-1899},
	doi = {10.1093/infdis/127.3.261},
	url = {https://doi.org/10.1093/infdis/127.3.261},
	author = {Levine, Myron M. and DuPont, Herbert L. and Formal, Samuel B. and Hornick, Richard B. and Takeuchi, Akio and Gangarosa, Eugene J. and Snyder, Merrill J. and Libonati, Joseph P.}
}
@article {ldfhtgsl73,
	title = {Pathogenesis of Shigella dysenteriae 1 (Shiga) Dysentery},
	journal = {Journal of Infectious Diseases},
	volume = {127},
	year = {1973},
	pages = {3},
	author = {Levine, M M and DuPont, H L and Formal, S B and Hornick, R B and Takeuchi, A and Gangarosa, E J and Snyder, M J and Libonati, J P}
}
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	number = {2},
	year = {2004},
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	url = {http://www.nature.com/nrmicro/journal/v2/n2/pdf/nrmicro818.pdf},
	author = {Kaper, J. B. and Nataro, J. P. and Mobley, H. L.}
}
@article {knm04,
	title = {Pathogenic Escherichia coli},
	journal = {Nature Reviews},
	volume = {2},
	number = {2},
	year = {2004},
	pages = {123-140},
	url = {http://www.nature.com/nrmicro/journal/v2/n2/pdf/nrmicro818.pdf},
	author = {Kaper, J B and Nataro, J P and Mobley, H L}
}
@article {knm04,
	title = {Pathogenic Escherichia coli. Nature Reviews},
	journal = {Microbiology.},
	volume = {2},
	number = {2},
	year = {2004},
	pages = {123-140},
	url = {http://www.nature.com/nrmicro/journal/v2/n2/full/nrmicro818.html},
	author = {Kaper, J. B. and Nataro, J. P. and Mobley, H. L.}
}
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	author = {Sabino, Raquel and Ver{\'\i}ssimo, Cristina and Cunha, Maria Ana and Wergikoski, Bela and Ferreira, Filipa C and Rodrigues, Raquel and Parada, Helena and Falc{\~a}o, Leonor and Rosado, Laura and Pinheiro, Catarina and others}
}
@article {bjcc90,
	title = {Pathogenicity of Acanthamoeba and Corynebacterium in the Rat Cornea},
	journal = {Archives of Ophthalmology},
	volume = {108},
	year = {1990},
	pages = {1},
	url = {https://jamanetwork.com/journals/jamaophthalmology/article-abstract/638228},
	author = {Badenoch, P R and Johnson, A M and Christy, P E and Coster, D J}
}
@article {dparpe08,
	title = {Pathogenicity of severe acute respiratory coronavirus deletion mutants in hACE-2 transgenic mice},
	journal = {Virology.},
	volume = {376},
	year = {2008},
	pages = {2},
	abstract = {<p>Recombinant severe acute respiratory virus (SARS-CoV) variants lacking the group specific genes 6, 7a, 7b, 8a, 8b and 9b (rSARS-CoV-Delta[6-9b]), the structural gene E (rSARS-CoV-DeltaE), and a combination of both sets of genes (rSARS-CoV-Delta[E,6-9b]) have been generated. All these viruses were rescued in monkey (Vero E6) cells and were also infectious for human (Huh-7, Huh7.5.1 and CaCo-2) cell lines and for transgenic (Tg) mice expressing the SARS-CoV receptor human angiotensin converting enzyme-2 (hACE-2), indicating that none of these proteins is essential for the viral cycle. Furthermore, in Vero E6 cells, all the viruses showed the formation of particles with the same morphology as the wt virus, indicating that these proteins do not have a high impact in the final morphology of the virions. Nevertheless, in the absence of E protein, release of virus particles efficacy was reduced. Viruses lacking E protein grew about 100-fold lower than the wt virus in lungs of Tg infected mice but did not grow in the brains of the same animals, in contrast to the rSARS-CoV-Delta[6-9b] virus, which grew almost as well as the wt in both tissues. Viruses lacking E protein were highly attenuated in the highly sensitive hACE-2 Tg mice, in contrast to the minimal rSARS-CoV-Delta[6-9b] and wt viruses. These data indicate that E gene might be a virulence factor influencing replication level, tissue tropism and pathogenicity of SARS-CoV, suggesting that DeltaE attenuated viruses are promising vaccine candidates.</p>
},
	doi = {10.1016/j.virol.2008.03.005},
	url = {https://www.sciencedirect.com/science/article/pii/S004268220800175X},
	author = {DeDiego, M L and Pewe, L and Alvarez, E and Rejas, M T and Perlman, S and Enjuanes, L}
}
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	pages = {379{\textendash}389},
	author = {DeDiego, Marta L and Pewe, Lecia and Alvarez, Enrique and Rejas, Maria Teresa and Perlman, Stanley and Enjuanes, Luis}
}
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	year = {Submitted},
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	author = {CDC}
}
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	year = {1986},
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	author = {Christie, AB}
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	year = {2008},
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	year = {2009},
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	url = {http://www.ajtmh.org.proxy1.cl.msu.edu/content/80/5/812.full.pdf+html},
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}
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	title = {Postexposure prophylaxis against anthrax: Evaluation of various treatment regimes in intranasally infected guinea pigs},
	journal = {Infection and Immunity},
	volume = {70},
	number = {1},
	year = {2002},
	pages = {6231-6241},
	url = {http://www.ncbi.nlm.nih.gov/pmc/articles/PMC130326/},
	author = {Altboum, Z. and Gozes, Y. and Barnea, A. and Pass, A. and White, M. and Kobiler, D.}
}
@article {agbpwk02,
	title = {Postexposure Prophylaxis against Anthrax: Evaluation of Various Treatment Regimens in Intranasally Infected Guinea Pigs},
	journal = {Infection and Immunity},
	volume = {70},
	year = {2002},
	pages = {11},
	author = {Altboum, Z. and Gozes, Y. and Barnea, A. and Pass, A. and White, M. and Kobiler, D.}
}
@article {altboum2002postexposure,
	title = {Postexposure prophylaxis against anthrax: evaluation of various treatment regimens in intranasally infected guinea pigs},
	journal = {Infection and immunity},
	volume = {70},
	number = {11},
	year = {2002},
	pages = {6231{\textendash}6241},
	publisher = {Am Soc Microbiol},
	author = {Altboum, Zeev and Gozes, Yehoshua and Barnea, Ada and Pass, Avi and White, Moshe and Kobiler, David}
}
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	year = {2009},
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	author = {Akinyemi, Kabir O and Atapu, Audu D and Adetona, Olabisi O and Coker, Akitoye O}
}
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}
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}
@article {conlon_predictors_1996,
	title = {Predictors of prognosis and risk of acute renal failure in patients with {Rocky} {Mountain} spotted fever},
	journal = {The American Journal of Medicine},
	volume = {101},
	number = {6},
	year = {1996},
	note = {Publisher: Elsevier},
	pages = {621{\textendash}626},
	abstract = {{\textless}h3{\textgreater}Background{\textless}/h3{\textgreater}{\textless}p{\textgreater}Acute renal failure has long been associated with severe Rocky Mountain spotted fever (RMSF). Despite many descriptions of the protean manifestations of this disease, relatively little is known concerning the risk factors for acute renal failure. Only a few studies have examined the outcome of patients infected with \textit{Rickettsia rickettsii} who develop renal insufficiency, and these studies had methodological problems.{\textless}/p{\textgreater}{\textless}h3{\textgreater}Objective{\textless}/h3{\textgreater}{\textless}p{\textgreater}To study the incidence, risk factors, and outcomes of acute renal failure in a large group of hospitalized patients with definite or probable RMSF.{\textless}/p{\textgreater}{\textless}h3{\textgreater}Methods{\textless}/h3{\textgreater}{\textless}p{\textgreater}The clinical records of 114 patients with definite or probable RMSF were retrospectively reviewed to identify clinical and biochemical abnormalities at the time of admission that were associated with the development of acute renal failure and subsequent mortality. Renal failure was defined as a serum creatinine (Cr) above 2 mg/dL. Logistic regression was used to study the association between these variables and the outcomes during hospitalization: death and the development of acute renal failure.{\textless}/p{\textgreater}{\textless}h3{\textgreater}Results{\textless}/h3{\textgreater}{\textless}p{\textgreater}The mortality rate in this series was 14\%; 19\% of the patients developed acute renal failure. The development of acute renal failure increased the odds ratio (OR) of dying by a factor of 17 (\textit{P} = 0.001). Factors at the time of hospitalization that were associated at a univariate level with subsequent mortality included elevated serum Cr, increased age, increased level of AST, increased level of bilirubin, decreased serum sodium and platelet count, the presence of neurological involvement, and being male. Both the presence of neurological involvement and an elevated serum Cr at presentation were independently associated with increased mortality by multivariate analysis. Three patients developed acute renal failure that required hemodialysis, and only 1 of these 3 patients survived; he was ultimately discharged with a normal serum Cr. Factors at presentation that were associated with the development of acute renal failure included increased bilirubin, increasing age, thrombocytopenia, and the presence of neurological involvement. Both age and decreased platelet count at presentation were independently associated with the development of acute renal failure by multivariate analysis.{\textless}/p{\textgreater}{\textless}h3{\textgreater}Conclusion{\textless}/h3{\textgreater}{\textless}p{\textgreater}Acute renal failure was a frequent complication of RMSF in this series of patients from a tertiary referral medical center. The presence of acute renal failure was strongly associated with death. Clinical and biochemical variables are useful in predicting which patients will develop acute renal failure.{\textless}/p{\textgreater}},
	issn = {0002-9343, 1555-7162},
	doi = {10.1016/S0002-9343(96)00332-4},
	url = {https://www.amjmed.com/article/S0002-9343(96)00332-4/abstract},
	author = {Conlon, Peter J. and Procop, Gary W. and Fowler, Vance and Eloubeidi, Mohamed Ali and Smith, Stephen R. and Sexton, Daniel J.}
}
@article {c96,
	title = {Predictors of prognosis and risk of acute renal failure in patients with Rocky Mountain spotted fever},
	journal = {The American journal of medicine},
	volume = {101},
	number = {6},
	year = {1996},
	pages = {621-626},
	author = {Conlon, Peter J}
}
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	year = {2015},
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	url = { https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4535930/ },
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}
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}
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	url = {http://imsear.hellis.org/handle/123456789/542},
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}
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	author = {Leoni, E and Legnani, PP and Sabattini, MA Bucci and Righi, F}
}
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	organization = {87-99},
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}
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}
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}
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}
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	author = {Zichichi, Leonardo and Asta, Gaetano and Noto, Giuseppe}
}
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}
